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- W2320632559 abstract "Background: Hepatic stellate cells (HSC) are the major source for extracellular matrix (ECM) production in liver fibrosis. Endoglin (ENG) is a type III auxiliary receptor for TGF-β that is expressed on HSCs. Due to different splicing there are two ENG isoforms, L- and S-Endoglin, which modulate TGF-β signaling differentially. TGF-β is the a profibrotic cytokine expressed in response to liver injury. This study analyzes the role of ENG and TGF-β signaling in two models of experimental liver fibrosis by cell line specific endoglin deletion in HSCs." @default.
- W2320632559 created "2016-06-24" @default.
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- W2320632559 date "2014-01-13" @default.
- W2320632559 modified "2023-10-13" @default.
- W2320632559 title "Hepatic stellate cell specific endoglin deletion modulates TGF-β signaling and aggravates liver fibrosis in two experimental models of murine liver injury" @default.
- W2320632559 doi "https://doi.org/10.1055/s-0033-1360866" @default.
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