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- W2321088225 abstract "Extracellular stimuli evoke the synthesis of intracellular second messengers, several of which couple to the release of Ca(2+)from Ca(2+)-storing organelles via activation of cognate organellar Ca(2+)-channel complexes. The archetype is the inositol 1,4,5-trisphosphate (IP3) and IP3receptor (IP3R) on the endoplasmic reticulum (ER). A less understood, parallel Ca(2+)signalling cascade is that involving the messenger nicotinic acid adenine dinucleotide phosphate (NAADP) that couples to Ca(2+)release from acidic Ca(2+)stores [e.g. endo-lysosomes, secretory vesicles, lysosome-related organelles (LROs)]. NAADP-induced Ca(2+)release absolutely requires organellar TPCs (two-pore channels). This review discusses how ER and acidic Ca(2+)stores physically and functionally interact to generate and shape global and local Ca(2+)signals, with particular emphasis on the two-way dialogue between these two organelles." @default.
- W2321088225 created "2016-06-24" @default.
- W2321088225 creator A5010531351 @default.
- W2321088225 date "2016-04-11" @default.
- W2321088225 modified "2023-10-18" @default.
- W2321088225 title "Ca2+ dialogue between acidic vesicles and ER" @default.
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- W2321088225 doi "https://doi.org/10.1042/bst20150290" @default.
- W2321088225 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27068968" @default.
- W2321088225 hasPublicationYear "2016" @default.
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