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- W2321839546 abstract "The metabolic action of 2-4-dinitrophenol has been the subject of several recent and informative studies. Thus the greatly increased oxygen consumption characteristic of its administration to animals and man seems from the observations of Hall, Tainter, Cutting and coworkers1,2 to be largely at the expense of fat as a fuel, at least when the stimulus augmenting metabolism is not too abrupt and overwhelming. There is general agreement that dinitrophenol does not increase the output of nitrogen in the urine1-5 and hence protein catabolism seems to be little affected. The injection of large and generally fatal doses into animals causes some elevation of the blood sugar and a considerable loss of liver and muscle glycogen, but smaller doses have little effect on the blood sugar.2,6,7 The glycemic effect of large doses of dinitrophenol is not dependent upon asphyxia8 and examination of the blood shows no very significant shift in the acid-base balance.2,8,9 Studies of the ammonia, organic acid and ketone body ..." @default.
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- W2321839546 date "1937-10-01" @default.
- W2321839546 modified "2023-09-27" @default.
- W2321839546 title "Effect of Dinitrophenol on Experimental Diabetes" @default.
- W2321839546 doi "https://doi.org/10.3181/00379727-37-9431" @default.
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