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- W2326410366 abstract "Amac: Koroner arter hastaligi (KAH), hem genetik hem de cevresel faktorlerden etkilenen cok faktorlu bir hastaliktir. Aday genlerdeki tek nukleotid polimorfizmleri (SNP) bu tur multifaktoryel hastaliklara yatkinliga neden olurlar. Bu yuzden, KAH etiyopatogenezinde rol oynayan genlerde SNPlerin arastirilmasi, onemli hale gelir. Bu calismada, KAH olusumu uzerinde, matriks metalloproteinaz-9 (MMP9) -1562 C/T ve metilentetrahidrofolat reduktaz (MTHFR) 677 C/T polimorfizmlerinin bagimsiz ve sinerjistik etkileri arastirildi.Yontemler: 109 koroner arter hastasi ve 108 saglikli kontrol olmak uzere toplam 217 birey incelendi. MTHFR 677 C/T ve MMP-9 -1562 C/T polimorfizmleri icin genotipler polimeraz zincir reaksiyonu (PCR)- restriksiyon fragmani uzunluk polimorfizmi (RFLP) ile belirlendi.Bulgular: KAH ile MMP-9 -1562 C/T ve MTHFR - 677 C/T polimorfizmlerinin genotipleri ve alel frekanslari arasinda istatistiksel olarak anlamli bir farklilik olmadigi bulundu. (p> 0.05) MMP9 -1562 C/T polimorfizmi icin TT homozigot genotipi hic bir grupta bulunmadi. Bununla birlikte, Guneydogu Anadolu Bolgesinde C aleli ve CC genotipi her iki polimorfizm icin hakim iken, TT genotipi ise cok nadir olarak bulundu. Sonuc: MTHFR 677 C/T ve MMP9 -1562 C/T polimorfizmleri ile koroner arter hastaligi arasinda iliski bulunmadi. Ancak Guneydogu Anadolu bolgesinde TT genotipinin cok nadir oldugu belirlendi" @default.
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- W2326410366 date "2016-03-01" @default.
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- W2326410366 title "MTHFR and MMP-9 Genetic Variants in Coronary Artery Disease" @default.
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- W2326410366 doi "https://doi.org/10.5798/diclemedj.0921.2016.01.0637" @default.
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