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- W2326519219 abstract "代謝抑制による心室筋活動電位の短縮をみた後, 再酸素化すると活動電位持続時間 (APD) の―過性延長 (rebound現象) を認めた.この現象に対する代謝抑制の程度ならびに各種薬剤効果 (verapamil, nioorandil, glibenclamide, lanthanum; La, hexamethylene-amiloride; HMA) を検討した.犬心室筋を虚血類似液で20分間灌流後, 正常Tyrode液にて再酸素化を行なった.APDは再酸素化後, 代謝抑制前に比し平均21.0±7.3%延長し, その程度は代謝抑制の程度に依存した.Verapamil, nicorandilを代謝抑制前から灌流するとrebound現象は抑制された.Verapamilを再酸素化の時点以後に加えても, rebound現象に変化はみられず, LaまたはHMA添加により抑制された.以上より, この現象の機序として, 代謝抑制時の細胞内カルシウム蓄積, ならびに再酸素化後の細胞内力ルシウム濃度やpHの変化が関与していることが示唆された." @default.
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- W2326519219 date "1994-01-01" @default.
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- W2326519219 title "On the transient prolongation of cardiac action potential duration after metabolic inhibition." @default.
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- W2326519219 doi "https://doi.org/10.5105/jse.14.242" @default.
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