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- W233059216 endingPage "46" @default.
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- W233059216 abstract "Publisher Summary It is unfeasible and unnecessary to coerce the complex biological events contributing to the origin of resistance into a single theory. Experimental evidence suggests that a few models define most examples of resistance development, particularly if the models are so combined as to provide either genotypic or phenotypic alterations of resistance level. If the body of present knowledge is to be kept sufficiently resilient to absorb new information without the impairment of established principles, it must be granted that resistance can arise in a variety of ways to be considered as complementary rather than mutually exclusive. This does not deny that a relatively important place must be assigned to certain methods of resistance development. Geneticists have been concerned with the spontaneous origin of resistance by mutation and the subsequent establishment of mutant clones by selective action of the drug." @default.
- W233059216 created "2016-06-24" @default.
- W233059216 creator A5005626461 @default.
- W233059216 creator A5044704219 @default.
- W233059216 date "1955-01-01" @default.
- W233059216 modified "2023-10-06" @default.
- W233059216 title "Microbial Drug Resistance" @default.
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- W233059216 doi "https://doi.org/10.1016/s0065-2660(08)60092-9" @default.
- W233059216 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/13258371" @default.
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