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- W2331271513 endingPage "1534" @default.
- W2331271513 startingPage "1524" @default.
- W2331271513 abstract "Telomeres help maintain genome integrity by protecting natural chromosome ends from being recognized as damaged DNA. When telomeres become dysfunctional, they limit replicative lifespan and prevent outgrowth of potentially cancerous cells by activating a DNA damage response that forces cells into senescence or apoptosis. On the other hand, chromosome ends devoid of proper telomere protection are subject to DNA repair activities that cause end-to-end fusions and, when cells divide, extensive genomic instability that can promote cancer. While telomeres represent unique chromatin structures with important roles in cancer and aging, we have limited understanding of the way telomeres and the response to their malfunction are controlled at the level of chromatin. Accumulating evidence indicates that different types of posttranslational modifications act in both telomere maintenance and the response to telomere uncapping. Here, we discuss the latest insights on posttranslational control of telomeric chromatin, with emphasis on ubiquitylation and SUMOylation events." @default.
- W2331271513 created "2016-06-24" @default.
- W2331271513 creator A5023690208 @default.
- W2331271513 creator A5064778775 @default.
- W2331271513 date "2012-04-15" @default.
- W2331271513 modified "2023-10-12" @default.
- W2331271513 title "Posttranslational control of telomere maintenance and the telomere damage response" @default.
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