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- W2332682783 abstract "Acute pancreatitis represents an acute inflammation of the pancreas, which occurs as a result of failure of protective physiological mechanisms and premature activation of proteolytic proenzymes. The occurrence, severity and outcome of this disease are, to a certain extent, affected by genetic variations. The most important polymorphic proteins, with a potential of impact on the development and course of acute pancreatitis, include (a) pancreatic proteins: cationic trypsinogen (PRSS1), pancreatic secretory trypsin inhibitor (SPINK1) and cystic fibrosis transmembrane conductance regulator (CFTR), and (b) mediators of inflammation: tumor necrosis factor α (TNF-α) and β (TNF-β), interleukins 8 (IL-8) and 10 (IL-10), monocyte chemotactic protein-1 (MCP-1), glutathione-S-transpherasetheta-1 (GSTT-1), heat shock protein 70-2 (HSP70-2) and epithelial-derived human β-defensin (DEFB1). Previous investigations that described the important role of genetics in the occurrence and development of acute pancreatitis, suggest that genotyping as a permanent analysis can indicate the population at risk of developing the disease, serve as a marker of expected disease severity and provide clinical guidelines for timely and optimal treatment." @default.
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- W2332682783 date "2011-01-01" @default.
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- W2332682783 title "Influence of genetic polymorphism on the course and outcome of acute pancreatitis" @default.
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- W2332682783 doi "https://doi.org/10.5937/racter1102017r" @default.
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