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• W2334090220 abstract "Oxidative events ‐ the oxidation of low-density lipoproteins (LDL) in the first place ‐ have been shown to participate in the progression of atherosclerosis and precipitation of cardiovascular events. This occurs through modulation of important components of lesions of the vessel wall (smooth muscle cell proliferation, protease/antiprotease balance, endothelial functions). Our recent studies have provided evidence that the enzyme gamma-glutamyltransferase (GGT), normally found in serum, is often accumulated within the plaque environment in substantial amounts, and that this activity is a potential source of a variety of prooxidant species. Concurrently, epidemiological research has conclusively documented that the serum levels of GGT are an independent factor in prognosis of myocardial infarction and stroke in atherosclerotic patients. Several signs suggest that the GGT appearing in plaque tissue may originate from the serum enzyme, which in facts associates with the circulating lipoprotein fractions. Thus, data seem to point out that pathogenesis of atherosclerosis and in particular of the events leading to progression of the disease and acute cardiovascular events ‐ might include an as yet unexplored pathway, based on the prooxidant effects of gamma-glutamyltransferase accumulating as a result of LDL entry in the vessel wall." @default.
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• W2334090220 date "2008-01-01" @default.
• W2334090220 modified "2023-10-06" @default.
• W2334090220 title "A NOVEL MARKER OF PROGRESSION FOR ATHEROSCLEROSIS AND CARDIOVASCULAR DISEASES: SERUM GAMMA-GLUTAMYLTRANS- FERASE ENZYME ACTIVITY" @default.
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