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- W2334888454 endingPage "94" @default.
- W2334888454 startingPage "87" @default.
- W2334888454 abstract "How the lens ages successfully is a lesson in biological adaption and the emergent properties of its complement of cells and proteins. This living tissue contains some of the oldest proteins in our bodies and yet they remain functional for decades, despite exposure to UV light, to reactive oxygen species and all the other hazards to protein function. This remarkable feat is achieved by a shrewd investment in very stable proteins as lens crystallins, by providing a reservoir of ATP-independent protein chaperones unequalled by any other tissue and by an oxidation-resistant environment. In addition, glutathione, a free radical scavenger, is present in mM concentrations and the plasma membranes contain oxidation-resistant sphingolipids what compromises lens function as it ages? In this review, we examine the role of small molecules in the prevention or causation of cataracts, including those associated with diet, metabolic pathways and drug therapy (steroids)." @default.
- W2334888454 created "2016-06-24" @default.
- W2334888454 creator A5006497097 @default.
- W2334888454 creator A5011003263 @default.
- W2334888454 date "2017-03-01" @default.
- W2334888454 modified "2023-10-17" @default.
- W2334888454 title "Small molecules, both dietary and endogenous, influence the onset of lens cataracts" @default.
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