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• W2335825983 abstract "G Proteins couple receptors for diverse extracellular signals to effecters such as enzymes and ion channels. The majority of polypeptide hormones, all monoamine neurotransmitters, PGs, and even extracellular Ca’+ signal their target cells through G protein-coupled receptors (GPCR). In the past few years, mutations in G proteins and GPCR have been identified as the causes of several endocrine diseases. Understanding the molecular basis of these diseases provides valuable insights into G protein and GPCR structure and function, and may be important for diagnosis and treatment. In this paper, I briefly describe G protein-coupled signal transduction, provide a general framework for understanding how G protein and GPCR mutations cause endocrine disease, review specific endocrine disorders caused by G protein and GPCR mutations, and speculate on implications of this work for the etiology, diagnosis, and treatment of endocrine disorders." @default.
• W2335825983 created "2016-06-24" @default.
• W2335825983 creator A5004120768 @default.
• W2335825983 date "1996-01-01" @default.
• W2335825983 modified "2023-09-27" @default.
• W2335825983 title "GENETIC BASIS OF ENDOCRINE DISEASE Mutations in G Proteins and G Protein-Coupled Receptors in Endocrine Disease" @default.
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• W2335825983 hasPublicationYear "1996" @default.
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