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• W2336715872 abstract "This study was designed to determine whether oxy­ gen-derived free radicals play a role in the patho­ genesis of gastric lesions produced by hypotensive ischemia in the rat. To achieve this goal, allopuri­ nol, an inhibitor of xanthine oxidase (the enzyme responsible for the formation of superoxide radi­ cals); superoxide dismutase, a scavenger of superox­ ide radicals (02 -); and dimethyl sulfoxide, a scav­ enger of hydroxyl radicals (OH' ) were used. In the anesthetized rat, HCl (0.1 N) was instilled into the pylorus-ligated stomach, and the rat was bled to reduce the blood pressure to <30 mmHg. The blood pressure was maintained at <30 mmHg for 20 min and then the shed blood was retransfused. Twenty minutes after the retransfusion the rat was killed, the stomach was removed, and the area of gastric mucosal lesions was measured. Both allopurinol and superoxide dismutase, but not dimethyl sulfox­ ide, significantly protected against hemorrhagic shock-induced gastric lesions. These findings sug­ gest that oxygen-derived free radicals, particularly O2 -, play an important role in the formation of gastric lesions produced by ischemia plus HCl. Numerous experimental studies have demonstrated the association between hemorrhagic hypotension and the formation of gastric mucosal lesions. Hemor­ rhagic hypotension produces a reduction in gastric mucosal blood flow. Although a number of hypothe­ ses have been proposed to explain the mechanism of" @default.
• W2336715872 created "2016-06-24" @default.
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• W2336715872 date "1985-01-01" @default.
• W2336715872 modified "2023-09-27" @default.
• W2336715872 title "Role of Oxygen-Derived Free Radicals iJl Hemorrhagic Sh()ck-Induced Gastric Lesions in the Rat" @default.
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• W2336715872 hasPublicationYear "1985" @default.
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