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- W2339916985 abstract "The evidence that nicotine may cause or aggravate cardiovascular disease is reviewed. Nicotine may contribute to accelerated atherogenesis by inducing hyperlipidemia, injuring endothelial cells and/or promoting thrombosis, although the evidence is not conclusive. Nicotine is likely to contribute to acute ischemic events in people who already have coronary heart disease via adverse effects on systemic hemodynamics, by promoting thrombosis, constricting coronary arteries and/or facilitating arrhythmogenesis. Pharmacodynamic studies suggest that nicotine inhaled in cigarette smoke may have different cardiovascular effects than that absorbed more slowly, such as from nicotine gum or transdermal nicotine. The safety of chronic nicotine exposure, such as with medicinal use of nicotine, cannot be predicted and requires empiric evaluation." @default.
- W2339916985 created "2016-06-24" @default.
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- W2339916985 date "1991-01-01" @default.
- W2339916985 modified "2023-09-27" @default.
- W2339916985 title "Nicotine and Cardiovascular Disease" @default.
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- W2339916985 doi "https://doi.org/10.1007/978-3-0348-7457-1_74" @default.
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