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• W2340264230 abstract "Store-operated Ca 2+ channel (SOC) is defined as a channel that opens in response to depletion of the internal Ca 2+ stores. During the last decade, many investigators have made a great effort to identify and characterize SOC, and to evaluate its physiologic function and pathophysiologic relevance in a variety of cell lines, primary cultures, and native tissues. To date, accumulating evidence has demonstrated that SOC is an essential Ca 2+ entry mechanism in vascular smooth-muscle cells of renal microvasculature and glomerular mesangial cells, both of which tightly control glomerular hemodynamics and filtration. Store-operated Ca 2+ , combined with other types of Ca 2+ entry channels, constitutes a profile of Ca 2+ changes in response to physiologic vasoconstrictors and, thereby, regulates renal microcirculation and mesangial function. In addition, SOC is associated with altered Ca 2+ signaling occurring in diseased kidneys, such as diabetic nephropathy. Although the gating mechanism and molecular identity of SOC are still enigmatic and may be cell-type and tissue specific, data from several independent groups suggest that protein kinase C plays an important role in SOC activation and that certain isoforms of canonical transient receptor potential (TRPC) proteins are candidates of SOC in renal mlcrovessels and mesangial cells." @default.
• W2340264230 created "2016-06-24" @default.
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• W2340264230 date "2006-02-01" @default.
• W2340264230 modified "2023-10-15" @default.
• W2340264230 title "Store-Operated Ca²⁺ Channel in Renal Microcirculation and Glomeruli" @default.
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• W2340264230 doi "https://doi.org/10.1177/153537020623100204" @default.
• W2340264230 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16446490" @default.
• W2340264230 hasPublicationYear "2006" @default.
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