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- W2340384134 abstract "Ammonium intoxication in dogs was induced by intravenous administration of urease. This method affords a unique preparation for the study of ammonia metabolism in vivo, since, within limits, a self-perpetuating, cyclic release of ammonia is achieved. Through the action of the enzyme, NH 3 —N is released from urea to be resynthesized via the ornithine-arginine cycle, operating in liver, to urea, from which in turn the ammonia nitrogen is released. Measurements of blood NH 3 —N, plasma NH 3 —N, BUN and plasma glutamine before and at 5, 30, and 60 minutes post intoxication show a highly significant increase in plasma glutamine and NH 3 —N levels and a decrease in BUN following ammonium intoxication. In showing that glutamine synthesis plays a major role in the clearance of an overload of NH 3 —N from the blood, and presumably other tissues, these results are in good agreement with the findings of other investigators." @default.
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- W2340384134 date "1961-07-01" @default.
- W2340384134 modified "2023-09-26" @default.
- W2340384134 title "Urease poisoning in the dog" @default.
- W2340384134 doi "https://doi.org/10.1152/ajplegacy.1961.201.1.71" @default.
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