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• W2340765907 abstract "Background/Aims: Gastric mucosa phospholipids play an important protective role against exogenous and endogenous toxic agents. Recently, we described a significant alteration of phospholipid profile in patients with chronic atrophic gastritis without Helicobacter pylori infection. The aim of the present study was to assess the phospholipid composition of gastric biopsy specimens in 41 subjects with chronic gastritis in relation to H. py/ori infection (no. 26) and nonalcoholic cirrhosis (no. 18). Methods: Phospholipids were extracted from homogenate mucosal samples using Folch’s method, purified, and separated by thin-layer chromatography, while bound fatty acids were analyzed by gas liquid chromatography. Results: The amounts of five gastric phospholipid classes, their rank order, and percent distribution of the principal ones (phosphatidylcholine [PC] 58%, phosphatidylethanolamine [PE] 26%, and phosphatidylinositol 11% vs. values of 49, 19, and 14, respectively, in the earlier study) were confirmed in chronic gastritis without H. py/ori infection. H. pylori infection induced a dramatic reduction (about 30%) in the absolute amount of total phospholip ids (24.2 pg/mg protein versus 35.1 of the H. py/orinegative group; P < O.Ol), PC and PE being the most affected (-36% and -26%, respectively), while bound fatty acids remained unchanged. There was no difference in cirrhotic vs. noncirrhotic subjects. Conclusibns: (1) The development of gastritis is characterized by an alteration of the lipid mucosal pattern that can change with the different etiologies, the most dramatic varia tions being observed in the presence of H. py/ori infection; and (2) cirrhosis does not affect further the alteration in the phospholipid profile of the antral mucosa caused by chronic gastritis." @default.
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• W2340765907 date "1994-01-01" @default.
• W2340765907 modified "2023-09-24" @default.
• W2340765907 title "Lipids of Human Gastric Mucosa: Effect Infection and Nonalcoholic Cirrhosis of Helicobacter pylori" @default.
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