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- W2341821133 abstract "1-4 The short chain peptides (22 to 47 amino acids) interfere with the function of potassium ion channels while long-chain peptides (59 to 76 amino acid residues) modify the channel gating properties of the sodium channel. 2 Other venoms identified include those that act on the calcium and chloride ion channels, hyaluronidases, lysozymes and phospholipases. 2 Many of the toxins act on ion channels that play an important role in maintaining resting membrane potential of excitable cells like neurons and myocytes. 1-4 They produce persistent depolarization of autonomic nerves with release of neurotransmitters from the adrenal medulla and parasympathetic and sympathetic nerve endings (the “autonomic storm”). These neurotransmitters are largely responsible for the toxic cardiovascular manifestations. 5 The toxin may also directly affect myocardial contractility and excitability. Central nervous system effects include irritability, muscle rigidity, altered consciousness and convulsions. 4 Scorpion envenomation following sting of the Indian red scorpion, Mesobuthus tumulus is a common emergency in several parts of rural India. This and all other venomous scorpions of clinical importance belong to the Buthidae family of scorpions, which are widely distributed in warmer parts of the old and new worlds. 1 Majority of deaths following scorpion envenomation in most par ts of the world occur due to cardiovascular dysfunction. 1 Neurological complications are more common than cardiovascular manifestations after sting by scorpions of the Centruroides species found in North America," @default.
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- W2341821133 date "2009-04-01" @default.
- W2341821133 modified "2023-09-29" @default.
- W2341821133 title "Management of scorpion envenomation: need for a standard treatment protocol using drugs and antivenom." @default.
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