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- W2342496683 abstract "Estrogens play a crucial role in the regulation of physiological and pathophysiological processes. These hormones act through specific receptors, most notably the canonical estrogen receptors α and β (ERα and ERβ) and their truncated forms as well as the G protein-coupled estrogen receptor (GPER). Several studies have shown that GPER is expressed in many normal and cancer cells, including those of the breast, endometrium, ovary, testis and lung. Hormonal imbalance is one possible cause of cancer development. An accumulating body of evidence indicates that GPER is involved in the regulation of cancer cell proliferation, migration and invasion, it may act as a mediator of microRNA, and is believed to modulate the inflammation associated with neoplastic transformation. Furthermore, used in various treatment regimens anti-estrogens such as tamoxifen, raloxifen and fulvestrant (ICI 182.780), antagonists/modulators of canonical estrogen receptors, were found to be GPER agonists. This review presents the current knowledge about the potential role of GPER in neoplastic transformation." @default.
- W2342496683 created "2016-06-24" @default.
- W2342496683 creator A5052006733 @default.
- W2342496683 creator A5054682822 @default.
- W2342496683 creator A5075326917 @default.
- W2342496683 date "2016-07-01" @default.
- W2342496683 modified "2023-10-17" @default.
- W2342496683 title "The G protein-coupled estrogen receptor as a modulator of neoplastic transformation" @default.
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