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- W2344433085 endingPage "94" @default.
- W2344433085 startingPage "63" @default.
- W2344433085 abstract "The developing lung is vulnerable to adverse conditions during prenatal and postnatal development; the peripheral lung, including alveoli and small conducting airways, is especially vulnerable. Owing to a limited capacity for repair, the functional effects of lung injury or altered development during fetal or early postnatal life may persist into later life, affecting lung function and disease vulnerability. A range of environmental factors can likely cause epigenetic alterations in key genes regulating lung development, thus contributing to persistent changes in lung structure and/or function. New experimental evidence supports the effectiveness of cell-based therapies to prevent or repair neonatal lung injury, and clinical trials are now underway in very preterm infants at risk of developing bronchopulmonary dysplasia.Keywords Development FetusAlveoliConducting airwaysEpitheliumSurfactant Lung function Lung hypoplasiaTobacco smokingFetal growth restrictionPreterm birthInflammationOxidative stressAlcoholRespiratory infectionNutrition Mesenchymal stromal cells" @default.
- W2344433085 created "2016-06-24" @default.
- W2344433085 creator A5045451689 @default.
- W2344433085 creator A5058049993 @default.
- W2344433085 creator A5086923376 @default.
- W2344433085 date "2015-01-01" @default.
- W2344433085 modified "2023-10-15" @default.
- W2344433085 title "Fetal and Neonatal Origins of Lung Disease" @default.
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