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- W2345016288 abstract "In this issue of Blood, Miyakawa et al show that platelets and protease-activated receptor (PAR)-4 contribute to acetaminophen (APAP)-induced liver damage. Using various strategies in a mouse model of APAP overdose, the authors demonstrate that platelets participate in the progression of liver damage, and that the direct thrombin inhibitor lepirudin and PAR-4 deficiency attenuate hepatotoxicity. These findings have the potential to help identify future therapeutic targets for APAP-induced hepatotoxicity." @default.
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- W2345016288 date "2015-10-08" @default.
- W2345016288 modified "2023-09-30" @default.
- W2345016288 title "Platelets mediate acetaminophen hepatotoxicity" @default.
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- W2345016288 doi "https://doi.org/10.1182/blood-2015-07-659516" @default.
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