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• W2346648290 startingPage "75" @default.
• W2346648290 abstract "A direct link has been elucidated between brassinosteroid function and perception, and sucrose partitioning and transport. Sucrose regulation and brassinosteroid signaling cross-talk at various levels, including the well-described regulation of transcriptional gene expression: BZR-like transcription factors link the signaling pathways. Since brassinosteroid responses depend on light quality and quantity, a light-dependent alternative pathway was postulated. Here, the focus is on post-translational events. Recent identification of sucrose transporter-interacting partners raises the question whether brassinosteroid and sugars jointly affect plant innate immunity and plant symbiotic interactions. Membrane permeability and sensitivity depends on the number of cell surface receptors and transporters. More than one endocytic route has been assigned to specific components, including brassinosteroid-receptors. The number of such proteins at the plasma membrane relies on endocytic recycling, internalization and/or degradation. Therefore, vesicular membrane trafficking is gaining considerable attention with regard to plant immunity. The organization of pattern recognition receptors (PRRs), other receptors or transporters in membrane microdomains participate in endocytosis and the formation of specific intracellular compartments, potentially impacting biotic interactions. This minireview focuses on post-translational events affecting the subcellular compartmentation of membrane proteins involved in signaling, transport, and defense, and on the cross-talk between brassinosteroid signals and sugar availability." @default.
• W2346648290 created "2016-06-24" @default.
• W2346648290 creator A5047035598 @default.
• W2346648290 date "2016-07-01" @default.
• W2346648290 modified "2023-10-16" @default.
• W2346648290 title "Review: Post-translational cross-talk between brassinosteroid and sucrose signaling" @default.
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• W2346648290 doi "https://doi.org/10.1016/j.plantsci.2016.04.012" @default.
• W2346648290 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27181949" @default.
• W2346648290 hasPublicationYear "2016" @default.
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