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- W2347053742 endingPage "303" @default.
- W2347053742 startingPage "285" @default.
- W2347053742 abstract "SummaryAlthough a successful vaccine against HBV has been implemented in 184 countries, eradication of hepatitis B virus (HBV) is still not on the horizon. There are over 240 million chronic carriers of HBV globally. The risk of developing chronic hepatitis ranges from >90% in newborns of hepatitis Be antigen (HBeAg)-positive mothers, 25%–35% in children under 5 years of age and 7.5% across the complete genome, HBV is classified phylogenetically into at least nine genotypes. With between ~4% and 8% intergroup nucleotide divergence, genotypes A–D, F, H and I are classified further into subgenotypes. HBV genotypes/subgenotypes may have distinct geographical distribution and can develop different mutations in the regions of the HBV genome that code for HBeAg. These differences can be related to the role of HBV genotypes to the natural history of infection and mode of transmission. Thus genotypes/subgenotypes of HBV can be responsible for the different natural history of infection and modes of transmission in children, found in various regions of the world, where different genotypes/subgenotypes prevail. Copyright © 2016 John Wiley & Sons, Ltd." @default.
- W2347053742 created "2016-06-24" @default.
- W2347053742 creator A5075901638 @default.
- W2347053742 date "2016-05-03" @default.
- W2347053742 modified "2023-10-12" @default.
- W2347053742 title "The clinical implications of hepatitis B virus genotypes and HBeAg in pediatrics" @default.
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