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- W2347331352 abstract "Objective To investigate injury induced by hypoxia reoxygenation impacts on the expression of ICAM-1 in hippocampal neuron cells in culture and the protective function of YinXinDaMo. Methods The SD rats were allocated to a normal culture group,hypoxia reoxygenation alone group(HR group),a hypoxia reoxygenation+YinXinDaMo group,6-hour reoxygenation following 1-hour hypoxia.The measurements of ICAM-1 levels,cytoplasmic free calcium([Ca2+]i),nitric oxide synthase(NOS),superoxide dismutase(SOD),malonyl dialdehyde(MDA) and reduced glutatione form(GSH) were recorded. Results When stimulated by hypoxia reoxygenation,the elevated levels of ICAM-1,i and MDA were obtained in hippocampal neuron cells,and SOD,GSH,NOS decreased significantly.YinXinDaMo could obviously improve the expression of ICAM-1 in hippocampal neuron cells with hypoxia reoxygenation,and also could inhibit the MDA,[Ca2+]i overload and the decrease of SOD,GSH,NOS.Conclusion Hypoxia reoxygenation directly or indirectly activated ICAM-1 to induce hippocampal neuron cells injury,but Ca overload,oxygen free radicals and NOS system are also involved in hypoxia/reoxygenation injury.YinXinDaMo directly or indirectly inhibits the mentioned above system to relieve the hypoxia reoxygenation injury." @default.
- W2347331352 created "2016-06-24" @default.
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- W2347331352 date "2010-01-01" @default.
- W2347331352 modified "2023-09-22" @default.
- W2347331352 title "The effects of hypoxia reoxygenation induced injury on the expression of ICAM-1 in hippocampal neuron cells in culture and the protective function of YinXinDaMo" @default.
- W2347331352 hasPublicationYear "2010" @default.
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