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- W23520400 endingPage "1385" @default.
- W23520400 startingPage "1375" @default.
- W23520400 abstract "Extensive research and scientific efforts have been focused on the elucidation of the pathobiology of cellular and axonal damage following traumatic brain injury (TBI). Conversely, few studies have specifically addressed the issue of synaptic dysfunction. Synaptic junction proteins may be involved in post-TBI alterations, leading to synaptic loss or disrupted plasticity. A Synapse Protein Database on synapse ontology identified 109 domains implicated in synaptic activities and over 5000 proteins, but few of these demonstrated to play a role in the synaptic dysfunction after TBI. These proteins are involved in neuroplasticity and neuromodulation and, most importantly, may be used as novel neuronal markers of TBI for specific intervention." @default.
- W23520400 created "2016-06-24" @default.
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- W23520400 date "2014-08-15" @default.
- W23520400 modified "2023-10-18" @default.
- W23520400 title "Alteration in Synaptic Junction Proteins following Traumatic Brain Injury" @default.
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