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- W2355695146 abstract "Objective To determine the protection and mechanism of salidroside on ischemia-reperfusion injury in isolated rat hearts.Methods Hearts were perfused in langendorff mode for 10 min,and then subjected global ischemia for 30 min,followed by a 120 min reperfusion period.Hemodynamic parameters were recorded,including: LVEDP,LVDP,+dP/dtmax,and-dP/dtmin.After the reperfusion,hearts were incubated in for 15 min with 1% triphenyl tetrazolium chloride by TTC staining of myocardial infarct size in the isolated rat hearts.At the end of the experiment,malondialdehyde(MDA),superoxidi dismutase(SOD),and nitric oxide(NO) levels were measured by enzymatic method.Results Compared with the control group,salidroside induced lower LVEDP,higher LVDP,+dP/dtmax,and +dP/dtmin.After the administration,hemodynamics in isolated rat hearts improved.The infarct size of salidroside(14.7±5.5)% was significantly lower than that of the control group(28.4±4.6)%(P=0.000 12).In addition,this agent could decrease the activities of CK and MDA,thus increasing the activities of SOD and NO.Conclusion Salidroside can protect ischemia-reperfusion injury in isolation,suggesting one possible mechanism of salidroside on cardioprotection through the suppression of oxidative stress." @default.
- W2355695146 created "2016-06-24" @default.
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- W2355695146 date "2010-01-01" @default.
- W2355695146 modified "2023-09-28" @default.
- W2355695146 title "Protective effect and its mechanism of salidroside on ischemia-reperfusion injury in isolated rat hearts" @default.
- W2355695146 hasPublicationYear "2010" @default.
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