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- W2356670840 abstract "Aim:To explore the expression of the nuclear factor-κB in human meningioma. The correlation of NF-κB with the clinic significance,necrosis,calcification and peritumoral edema was analyzed in human meningioma. Methods: The expression of NF-κB p65 of 50 human meningiomas and 10 human common cerebral dura maters used as control were examined by immuno histochemical method. Results: There is no expression of NF-κB p65 in the control cerebral dura mater tissues, but some are different level expressions of NF-κBp65 are found in the meningioma tissues. The differences of level expressions of NF-κBp65,EGF,EGFR between the normal dura and meningioma are significant( P 0.005). In the different type mieningioma, the expressions of NF-κBp65 are significant different( P 0.005). Moreover,there are different level expressions of NF-κBp65,EGF,EGFR in the meningioma tissues. The expression of NF-κB is related to the expression of EGF( r =0.4524, P =0.001,0.005),but not to the expression of EGFR( r =-0.2506, P 0.05),and the necrosis,calcification,peritumoral edema ( r =-0.27, P 0.05; r =-0.045, P =0.754; r =-0.043, P =0.769),in meningioma tissue. The expression of EGF is not related to the peritumoral edema in meningioma tissue too( P 0.05). Conclusion:The expression of NF-κB is anomaly in the meningiomas, and it is possible related to occurrence、proliferation of meningioma cell. The expression of NF-κB is not related to the necrosis,calcification,peritumoral edema in meningioma tissue. The expression of EGF is not related to the peritumoral edema in meningioma tissue too. As the still deeper research of NF-κB is accomplished, a new way of treatment of meningioma by medicines will be found." @default.
- W2356670840 created "2016-06-24" @default.
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- W2356670840 date "2005-01-01" @default.
- W2356670840 modified "2023-09-27" @default.
- W2356670840 title "Expression of Nuclear Factor-κB in Human Meningioma and the Correlation with the Clinic Significance" @default.
- W2356670840 hasPublicationYear "2005" @default.
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