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- W2357172167 abstract "Crohn's disease(CD) has traditionally been considered a prototypical T-helper-1(Th1) condition mediated by the interleukin-12/interferon-γ/tumor necrosis factor(TNF) cytokine axis and an atypical Th2-type responses mediated by the interleukin-4/ interleukin-5/ interleukin-13 cytokine axis is involved in the pathophysiology of ulcerative colitis(UC).Recent studies demonstrate IL-23/IL-17 axis may play a key role in the pathogenesis of IBD and may represent a target for therapeutic intervention for inflammatory bowel disease(IBD).IL-23 induces the differentiation of naive CD4+ T cells into highly pathogenic helper Th17 cells that produce IL-17,IL-6,and TNF-α and subsequent causes colitis." @default.
- W2357172167 created "2016-06-24" @default.
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- W2357172167 date "2008-01-01" @default.
- W2357172167 modified "2023-09-24" @default.
- W2357172167 title "The interleukin-23/17 axis and inflammatory bowel disease" @default.
- W2357172167 hasPublicationYear "2008" @default.
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