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- W2357274014 abstract "Objective: To discuss the effects of gap junction function and calcium mediated signal transduction system on nongenotoxic carcinogenesis. Methods:Using scrape loading dye transfer (SLDT) and fluo 3 tracer technique, the influence of three nongenotoxic carcinogens on gap junction function and concentration of intracellular free calcium in BALB/c 3T3 cells was detected. Results: All the three chemicals significantly inhibited the gap junction intercellular communication (GJIC) in a dose dependent maner. Microcystins and tetradecanoylphorbol acetatel (TPA) could induce the increase of intracellular free calcium, but phenbarbital sodium could not cause significant intracellular free calcium oscillation in the cell line in tested dosage. Conclusion: GJIC is the common active site of these three kinds of nongenotoxic carcinogens, and the calcium mediated signal transduction system may mainly have effects on TPA like carcinogens." @default.
- W2357274014 created "2016-06-24" @default.
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- W2357274014 date "1998-01-01" @default.
- W2357274014 modified "2023-09-25" @default.
- W2357274014 title "EFFECTS OF MICROCYSTINS, PHENOBARBITAL SODIUM AND TETRADECANOYLPHORBOL ACETATE ON GAP JUNCTION INTERCELLULAR COMMUNICATION AND INTROCELLULAR FREE Ca 2+ IN BALB/c 3T3 CELLS" @default.
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