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- W2359203044 abstract "Recently, the studies showed that carotid body (CB), a traditional peripheral chemoreceptor, also played role of an immune-signal sensor in the body. However, it is not clear that whether peripheral proinflammatory cytokine, such as interleukin-1β (IL-1β), will influence the process of hypoxia sensing in CB. In order to understand the influence of IL-1β on the chemoreceptive function of carotid body (CB), the effect of IL-1β on the experimental acute hypoxia-induced firings in carotid sinus nerve (CSN), the nerve innervates CB, was studied in the anaesthetized rat by using extracellular recording method. The results showed that experimental acute hypoxia (EAH) induced an increase of CSN discharge rate. Topical application of ATP (0.1 mmol/L) and/or Ach (0.5 mmol/L) on CB induced a similar increase in CSN discharge rate as well. Furthermore, topical application of IL-1β (40 ?滋g/L) under normoxia condition also increased the discharge rate of CSN, in consistent to our previous result. However, the increase of CSN discharge rate induced by hypoxia or ATP/ACh administration was not affected if IL-1β was given in advance. These results indicate IL-1β does not have a modulatory action on the EAH-induced firings in CSN, at least in the anaesthetized rat." @default.
- W2359203044 created "2016-06-24" @default.
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- W2359203044 date "2007-01-01" @default.
- W2359203044 modified "2023-09-27" @default.
- W2359203044 title "THE EFFECT OF IL-1β ON EXPERIMENTAL ACUTE HYPOXIA-INDUCED FIRING OF RAT CAROTID SINUS NERVE" @default.
- W2359203044 hasPublicationYear "2007" @default.
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