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- W2360712361 abstract "AIM: To study the protection of iptakalim hydrochloride (Ipt) against brain anoxia in mice. METHODS: The models of brain anoxia were made in mice after the administration of Ipt ( 0.5 , 2.0 , 8.0 mg·kg -1 , n=12, ig)for 3 wk. NS and vitamin E were set as controls. The glutathione proxidase (GSH PX) activity, superoxide dismutase (SOD) activity and the lipid peroxide (MDA) content were determined in the brain homogenates of models, and the values of erythrocyte membrane fluidity were measured by the fluorescence polarization technique with DPH fluorescence probe. RESULTS: Compared with the control groups, Ipt ( 0.5 , 2.0 , and 8.0 mg·kg -1 , ig) markedly prolonged the breathing time and increased the fluidity of erythrocyte membrane in a dwse dependent manner, but had no apparent effects on the activity of GSH PX and SOD. CONCLUSION: The protection of Ipt against brain anoxia is related to increasing the fluidity of erythrocyte membrane and decreasing blood viscosity." @default.
- W2360712361 created "2016-06-24" @default.
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- W2360712361 date "2004-01-01" @default.
- W2360712361 modified "2023-09-28" @default.
- W2360712361 title "Protection of iptakalim hydrochloride against brain anoxia in mice" @default.
- W2360712361 hasPublicationYear "2004" @default.
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