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- W2364114003 abstract "Huang-gushu showed marked power to counteract arrhythmias induced by aconitine in the models of rats and dogs. Arrhythmias due to the coronary artery ligation could be antagonized by the drug in the anaesthetized rats. Delayed outward K+ current and outwad tail current were specifically inhibited by Huang-gushs in a concentration dependent fashion.The drug inhibited IK more strongly as the membrane potential becoame more positive from 0 mV to + 60 mV. The value of IK was attenuated to 810 pA from 1160 pA of control and the value of tail current was reduced to 196 pA from 331 pA of control at 60 mV. The results stronply suggest that the principal mechanism of Huang-gushu in antiarrhythmic action is to inhibit voltage-dependent K+ current." @default.
- W2364114003 created "2016-06-24" @default.
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- W2364114003 date "1996-01-01" @default.
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- W2364114003 title "Anti-arrhythmic effect of Huang-gushu" @default.
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