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- W2364310565 abstract "Focal recruitment of monocytes and lymphocytes is one of the earlist detectable cellular responses in the formation of atherosclerotic lesions(AS). To investigate the role of some risk factors in pathogenesis of AS, human Umbilical vein endothelial cells(HUVEC) were exposed to mmLDL,rTNF or low oscillatory shear stress and then the assay of cells adhesion percent, expression of leukocyte adhesion molecular VCAM 1 ICAM 1 P selectin and nuclear translocation of NFκB in HUVEC were examined by cell immunochemistry and hybridization in situ. The results indicated that U937 adhesion to HUVEC exposed to mmLDL or γTNF were 7.0fold and 9.0fold respectively as that of control(both P 0.01). γTNF exposure induced hyperadherance by VCAM 1,ICAM 1,P selectin dependent mechanism. The cell surface expression of VCAM 1,ICAM 1in HUVEC was not increased after treatment with mmLDL. Low oscillory shear stress augments the expression of VCAM 1 both at protein and mRNA levels. Three risk factors elicited nuclear translocation of NFκB P65. The activation of endothelium NFκB by mmLDL, γTNF, low oscillory shear stress is a common signal pathway and may contribute to the formation of early atherosclerotic lesions." @default.
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- W2364310565 date "2002-01-01" @default.
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- W2364310565 title "Effect of some risk factors on adhesion molecular and NFκB activation in vascular endothelial cells" @default.
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