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- W2364473324 abstract "Hypertension is a major risk factor of cardiovascular disease, and the reduction of elevated blood pressure significantly reduced the risk of cardiovascular events. The pathophysiology of hypertension is complex and its the common process is increased bioavailability of reactive oxygen species(ROS), that is oxidative stress. In the cardiovascular system, ROS plays a physiological role in controlling endothelial function, vascular tone, and cardiac function, and a pathophysiological role in inflammation, hypertrophy,proliferation, migration, fibrosis, angiogenesis and so on, which also play roles in the endothelial dysfunction induced by hypertension.Although convincing data from animal studies support a causative role for oxidative stress in the pathogenesis of hypertension, there is no solid evidence that oxidative stress causes hypertension in humans yet. Endothelial dysfunction means reduced vasodilating response to endothelial stimuli. A large amount of evidences indicate that endothelial dysfunction is one of the mechanisms mediating the adverse vascular effects of hypertension. Hypertension-related endothelial dysfunction has been demonstrated to be the consequence of increased oxidative stress production in many studies.This review highlights the experimental studies and mechanism of oxidative stress and endothelial dysfunction in hypertension, and outlines the relationship between oxidative stress and endothelial dysfunction in the development of hypertension." @default.
- W2364473324 created "2016-06-24" @default.
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- W2364473324 date "2014-01-01" @default.
- W2364473324 modified "2023-09-23" @default.
- W2364473324 title "Oxidative Stress and Endothelial Dysfunction in Hypertension" @default.
- W2364473324 hasPublicationYear "2014" @default.
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