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- W2371041073 abstract "Objective In order to examine the effect of nicotinamide adenine dinucleotide(NADH) on electrophysiological remodeling(ER).Methods Twenty-four rabbits were randomly divided into four groups(control group,pacing/control group,pacing/NADH group,pacing/NADH+rotenone group).The hearts were removed and perfused by the Langendorff technique with continuous corresponding buffer,respectively.At the same time,pacing the right atrium was performed for 1 hour in all groups but not in control group.Before and after pacing/perfusing,atrial effective refractory period(AERP) was determined in all hearts.Then,right atrium tissue was obtained for measurement of calcium-handling proteins mRNA using semiquantitative reverse transcription-polymerase chain reaction(RT-PCR).Results After pacing 1 hour,AERP shortened significantly in pacing/control group and pacing/NADH+rotenone group,but in pacing/NADH group,AERP did not obviously change.The mRNA level of L-type Ca~(2+) channel decreased and the mRNA level of ryanodine receptor type 2 increased in pacing/control group and pacing/NADH+rotenone group,the mRNA level of sarcoplasmic reticulum calcium ATPase decreased in pacing/control group and held the line in pacing/NADH+rotenone group,but these changes were not found in pacing/NADH group.Conclusions The activation of NADH maybe one mechanism underlying short-term ER.[Chinese Journal of Cardiac Pacing and Electrophysiology,2006,20(1):66~68 ]" @default.
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- W2371041073 date "2006-01-01" @default.
- W2371041073 modified "2023-09-25" @default.
- W2371041073 title "The effect of nicotinamide adenine dinucleotide on electrical remodeling in atrial fibrillation." @default.
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