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- W2371667874 abstract "Objective To investigate the beneficial effects of atenolol on cardiomyocyte apoptosis after acute myocardial infarction (AMI)in rats. Methods AMI model was established with the ligation of anterior descending coronary artery in 59 randomly selected female SD rats. The 39 survivors were randomly assigned to AMI control (MI, n=18), Atenolol (10mg/kg/d) treatment (A,n=21) , groups with each being randomly reassigned to 48-hour and 4- week subgroups according to the time to observe, that is:MI48h(n=8) and MI4w(n=10) subgroups, A48h(n=11) and A4w (n=10)subgroups. Sham-opeated(S, n=10) group was selected randomly, and also reassigned to S48h(n=5) and S4w(n=5) subgroups. Cardiomyocyte apoptosis was detected with TUNEL. Results Compared with MI48h, M14w subgroup,no significantly change was found of myocyte apoptosis rate ( MAR) in infarction, border and non - in-farcted area(P0.05) in A48h subgroups; MAR was significantly decreased in the three areas mentioned above (P0.05-0.01) in A4w subgroups. Conclusion While treatment with atenolol, early after AMI in rats, atenolol could not reduce cardiomyocyte apoptosis in infarction area. After 4 weeks of treatment, atenolol could reduce cardionmycryteapoptosis in infarction/scar and border areas, and aternolol could also reduce cardiomycyteapoptosis in non - infarcted area." @default.
- W2371667874 created "2016-06-24" @default.
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- W2371667874 date "2005-01-01" @default.
- W2371667874 modified "2023-09-25" @default.
- W2371667874 title "The Beneficial Effects of Atenolol on Cardiomyocyteapoptosis after Acute Myocardial Infarction in Rats" @default.
- W2371667874 hasPublicationYear "2005" @default.
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