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- W2373508737 abstract "Aim: To establish and optimize the gastric ischemic postconditioning(GI-postc) model in rats,and preliminarily investigate the antioxidation mechanism of GI-postc.Methods:A total of 78 rats were randomly divided into 13 groups.Sham group and GI-R group were used as control.The experimental groups included four different R and I time groups (10,20,30,and 40 s R/I),four different cycle groups (1,3,5,and 10 cycles),and three different beginning time groups(1,2,and 4 min).Gastric mucosal injury index was detected.The content of malondialdehyde (MDA) and the activity of superoxidase dismutase (SOD) in gastric mucosa were detected.Results: GI-postc could significantly reduce gastric mucosal injury induced by I/R.There were significant differences among sham group,GI-R group,and four different R and I time groups(F=16.574,P0.001),among sham group,GI-R group,and four different cycle groups(F=34.815,P0.001),among sham group,GI-R group,and three different beginning time groups (F=48.499,P0.001).There were significant differences on MDA(F=27.250,31.450,and 27.520,P0.001) and SOD (F=72.662,57.220,and 50.650,P0.001) among these groups.Conclusion:The GI-postc model in rats has been established and optimized successfully." @default.
- W2373508737 created "2016-06-24" @default.
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- W2373508737 date "2011-01-01" @default.
- W2373508737 modified "2023-09-23" @default.
- W2373508737 title "Establishment of gastric ischemic postconditioning model in rats" @default.
- W2373508737 hasPublicationYear "2011" @default.
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