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- W2374059637 abstract "Objective To investigate the possible molecular pathogenesis of inflammatory reaction in the lung tissues ischemia -reperfusion(I/R)injury following hemorrhagic shock. Methods Rabbit model of ischemia - reperfusion injury following hemorrhagic shock wasused in this study. The blood air lung wet/dry(W/D) weight and contents of the tumor necrosis factor - α(TNF - α), interleukin - 6 (IL - 6),the neutrophils cell counts, albumin, C3a, C5a in bronchoalveolar lavage fluid (BALF)were analyzed. The pathological changes wereexamined with light microscope in lung tissues. The expressions of IKK - β and activation of NF - κB in the lung tissues were measured by insitu hybridization, immunohitochemistry combined with in situ quantitative analysis, respectively. Results In ischemia - reperfusion injurygroup, the expression of IKK - β mRNA, activation of NF - κB in lung tissues and the levels of all inspection guideline in BALF wereobviously higher than those of normal group, and the severity of lung tissue damages increased in ischemia - reperfusion(I/R)injury followinghemorrhagic shock. Conclusion Release of inflammatory cytokines induced by NF - κB signal transduction pathways might be an importantpathogenesis in the lung tissue ischemia - reperfusion(I/R)injury following hemorrhagic shock." @default.
- W2374059637 created "2016-06-24" @default.
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- W2374059637 date "2003-01-01" @default.
- W2374059637 modified "2023-09-25" @default.
- W2374059637 title "A study on mechanism or inflammatory pathology in the lung tissues ischemia - reperfusion injury following hemorrhagic shock" @default.
- W2374059637 hasPublicationYear "2003" @default.
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