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- W2374095910 abstract "Aim: To define the cellular mechanisms and pathophysiological properties of heart failure using transgenic Gαq and Gαq/ACⅥ mice.Methods:Action potential duration(APD),cardiac L-type calcium current(ICa,L)and transient outward K+ current(Ito)of the left ventricular myocytes isolated from Gαq, Gαq/ACⅥ transgenic mice and wild-type mice were examined using patch-clamp and electrophysiological techniques.Results: The myocytes isolated from Gαq mice showed prolonged APD50% and APD90% with reduced Ito.The above parameters in Gαq/ACⅥ mice were improved.Conclusion: Cardiac-directed overexpression of Gαq results in the left ventricular dysfunction,wherease increasing expression of cardiac ACⅥ makes significant improvement in the cardiac function." @default.
- W2374095910 created "2016-06-24" @default.
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- W2374095910 date "2007-01-01" @default.
- W2374095910 modified "2023-09-27" @default.
- W2374095910 title "Cardiac electrophysiological properties of transgenic Gαq and Gαq/ACVI mice" @default.
- W2374095910 hasPublicationYear "2007" @default.
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