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- W2374828075 abstract "Objective:To investigate whether the bone morphogenetic protein 13(BMP13) can promote C3H10T1/2 cell differentiation into cardiomyocytes in microenvironment and improve the cardiac function of the rats after myocardial infarct(MI).Methods:MI and sham operation were established in sprague dawley(SD) male rats.Totally 56 rats were divided into 4 groups: sham operation group(8 rats),MI group(16 rats),MI+GFP group(16 rats),and MI+BMP13 group(16 rats).Left ventricular(LV) function were assessed by echocardiography on day baseline and 4 weeks after operation.After measuring body weight once a week,on 4 weeks after completion of cardiac function test,rats were sacrificed to take the hearts.Differences in infarct sizes of hearts were compared by Masson's staining.Cardiac-specific structural protein α-MHC and cTnT were implanted in each group and detected by immunofluorescence assay.Results:Each C3H10T1/2 cells had about 80% of cells expressing green fluorescence.On 4 weeks after the experiment,heart function in the groups with cells implanted significantly improved,as compared with MI group,MI+BMP13 group more obvious(P0.05).Implanted cells in group MI+GFP and MI+BMP13 had cardiac cell-specific structural protein α-MHC and cTnT expression by immunofluorescence.Conclusion:BMP13 can induce differentiation of C3H10T1/2 cells into myocardial cells and improve cardiac function in myocardial infarction rats." @default.
- W2374828075 created "2016-06-24" @default.
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- W2374828075 date "2011-01-01" @default.
- W2374828075 modified "2023-09-24" @default.
- W2374828075 title "BMP13 on C_3H_(10)T_(1/2) cells to improve cardiac function in myocardial infarction rats" @default.
- W2374828075 hasPublicationYear "2011" @default.
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