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- W2375635605 abstract "Alzheimer's disease is a complex neurodegenerative disorder. A large number of genome-wide association studies have been performed, which have been supplemented more recently by the first epigenome-wide association studies, leading to the identification of a number of novel loci altered in disease. Twin studies have shown monozygotic twin discordance for Alzheimer's disease (Gatz et al., 2006), leading to the conclusion that a combination of genetic and epigenetic mechanisms is likely to be involved in disease etiology (Lunnon & Mill, 2013). This review focuses on identifying overlapping pathways between published genome-wide association studies and epigenome-wide association studies, highlighting dysfunctional synaptic, lipid metabolism, plasma membrane/cytoskeleton, mitochondrial, and immune cell activation pathways. Identifying common pathways altered in genetic and epigenetic studies will aid our understanding of disease mechanisms and identify potential novel targets for pharmacological intervention." @default.
- W2375635605 created "2016-06-24" @default.
- W2375635605 creator A5018086792 @default.
- W2375635605 creator A5049350976 @default.
- W2375635605 creator A5063525602 @default.
- W2375635605 creator A5074924251 @default.
- W2375635605 date "2016-06-01" @default.
- W2375635605 modified "2023-09-27" @default.
- W2375635605 title "Elucidating novel dysfunctional pathways in Alzheimer's disease by integrating loci identified in genetic and epigenetic studies" @default.
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