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- W2375682831 endingPage "205" @default.
- W2375682831 startingPage "198" @default.
- W2375682831 abstract "In response to luminal food stimuli during meals, enteroendocrine cells release gastrointestinal (GI) peptides that have long been known to control secretory and motor functions of the gut, pancreas and liver. Glucagon-like peptide-1 (GLP-1) has emerged as one of the most important GI peptides because of a combination of functions not previously ascribed to any other molecule. GLP-1 potentiates glucose-induced insulin secretion, suppresses glucagon release, slows gastric emptying and may serve as a satiation signal, although the physiological status of the latter function has not been fully established yet. Here we review the available evidence for intestinal GLP-1 to fulfill a number of established empirical criteria for assessing whether a hormone inhibits eating by eliciting physiological satiation in man and rodents." @default.
- W2375682831 created "2016-06-24" @default.
- W2375682831 creator A5010566523 @default.
- W2375682831 creator A5011289374 @default.
- W2375682831 creator A5038498547 @default.
- W2375682831 date "2015-08-28" @default.
- W2375682831 modified "2023-10-13" @default.
- W2375682831 title "Intestinal GLP-1 and satiation: from man to rodents and back" @default.
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