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- W2378588001 abstract "Apoptosis is involved in the pathological process of hypoxic-ischemic brain damage.Once hypoxia-ischemia occurs,a number of independent pathways can lead to apoptosis respectively.The mitochondrial pathway attached increasing attention.A variety of factors promoting and anti-apoptotic play roles in the mitochondria,triggering mitochondrial function and biochemical changes,and determining the fate of cells after injury.The key processs of mitochondrial pathways include the generation of apoptotic stimulator,changes in mitochondrial permeability,release of mitochondrial apoptotic factors,caspase family activation,and so on.If they can be blocked on the above processes,excessive apoptosis of neurons can be inhibited and the hypoxic-ischemic brain injury will be reduced.Clinical research and animal experiments have confirmed that hyperbaric oxygen therapy can reduce neuronal apoptosis.However,the mechanism is so complex that it is not yet completely clear.This article will explore the possible mechanism of hyperbaric oxygen therapy for mitochondrial pathways of apoptosis." @default.
- W2378588001 created "2016-06-24" @default.
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- W2378588001 date "2010-01-01" @default.
- W2378588001 modified "2023-09-25" @default.
- W2378588001 title "The Effect of Hyperbaric Oxygen on Hypoxic-Ischemic Neuronal Apoptosis Through Mitochondrial Pathway" @default.
- W2378588001 hasPublicationYear "2010" @default.
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