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- W2380455764 abstract "To investignte the changes of glutamate trareporter function, superoxide dismutase (SOD) achvity andmalondiadehyde (MDA) content in rats with injury of cerebral ischemia-reperfusion and antagonistical effect of dexanetha-sone. METHODS: A model of brain ischemia-reperfusion injury was estahlished by clipped three arteries and then releasedto repedese blood into brain in rats. The glutamte transporter funtions, SOD activities and MDA contents in cerebral con-tex, hippocampus and striatum of control, I-R and dexamethasone groups were detendned. RESULTS: Glutamate trans-porter function and SOD activity of ahave three sites in I-R group were decreased significanily (P 0.05, P 0.01 ) andMDA contents were incamd (P 0.05, P 0.01 ), comatal with control group. The glutamate transporter function,SOD achvity and MDA content in the ahave tae sites of I-R+dexamthasone group were recovered, compared with I-Rgroup. CONCLUSION: Oxygrn free radicals, which wele pnduced in the ischenda-repefuosion injury might result in theinhibihon of glutamate transporter. Dexamathasoe may antagonize the injury effect of the oxygen free radicals by acting onglutamate transporter in membrane." @default.
- W2380455764 created "2016-06-24" @default.
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- W2380455764 date "1999-01-01" @default.
- W2380455764 modified "2023-09-25" @default.
- W2380455764 title "Damage of glutamate transporter function during ischemia-reperfusion injury of rat brain and antagonistical effect of dexamethasone" @default.
- W2380455764 hasPublicationYear "1999" @default.
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