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- W2381076568 abstract "AIM: To investigate the influences of bypass-activated complement and TNF-α on intercellular adhesion molecule 1(ICAM-1) expression in endothelial cells, and polymorphonuclear neutrophil-endothelial cell (PMN-EC) adhesion. METHODS: In vitro, zymosan-activated human serum(ZAHS) and/or TNF-α directly stimulated the HUVECS monolayers. PMN-EC adhesion percentage was measured, and modified whole-cell ELISA was used for measurement of ICAM-1. RESULTS: ZAHS alone resulted in no significant change in the degree of PMN adhesion and the level of ICAM-1. Activation of HUVECS with TNF-α followed by ZAHS stimulation resulted in greater increase in PMN-EC adhesion and ICAM-1 expression, as compared with the activation with TNF-α alone. CONCLUSION: Bypass-activated complement can promote ICAM-1 expression and neutrophil -endothelium adhesion induced by TNF-α, and so it is effective for promoting inflammation." @default.
- W2381076568 created "2016-06-24" @default.
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- W2381076568 date "2003-01-01" @default.
- W2381076568 modified "2023-09-23" @default.
- W2381076568 title "Influences of bypass-activated complement and TNF-α on endothelial cells to express ICAM-1 and PMN-EC adhesion" @default.
- W2381076568 hasPublicationYear "2003" @default.
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