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- W2382006148 abstract "We systematically studied the effect of high sustained +Gz on the brain and its mechanism in past ten years by animal centrifuge experiments. On the basis of the facts we observed and the more recent advances in acceleration physiology, we put forward a compound mechanism hypothesis to offer a possible explanation for +Gz-induced brain injury and dysfunction of learning and memory. It states that, ischemia during high G exposure might be the main factor accounting for + Gz-induced brain injury and dysfunction of learning and memory, including transient depression of brain energy metabolism, disturbance of ion homeostasis, increased blood-brain barrier permeability, increased brain nitric oxide synthase and c-fos expression, and the protective effect of heat shock protein 70(HSP70). In addition, the large rapid change of intracranial pressure and increased stress during +Gz exposure, and the hemorrheologic change after +Gz exposure might be one of the important factors accounting for +Gz-induced brain injury and dysfunction of learning and memory." @default.
- W2382006148 created "2016-06-24" @default.
- W2382006148 creator A5030519526 @default.
- W2382006148 date "2004-01-01" @default.
- W2382006148 modified "2023-09-24" @default.
- W2382006148 title "Multiple Mechanism Hypothesis on + Gz-induced Brain Injury and Dysfunction of Learning and Memo- ry" @default.
- W2382006148 hasPublicationYear "2004" @default.
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