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- W2384385204 abstract "Objective: The pathological basis of infrapatellar fat pad injury is repeated injury, and impingement of the infrapatellar fat pad causing fat pad proliferation and inflamation. The model of infrapatellar fat pad and synovium injury was established, and the level of SP, CGRP and NPY were observed. Methods: We used pockets of water compressing rabbit infrapatellar fat pad, the pressure in pockets of water remaining 330～340mmHg, accompanied by knee joint flexion and extension 2 hours every day. Seven days later, 0.9% NaCl 0.5ml was injected into the joint cavity in control and injury group. The joint flushing fluid was then extracted and measured by radioimmunoassay. Results: SP: injury group 71.34±24.44 pg/ml, control group 21.91±14.44 pg/ml, ( P 0.01) ; CGRP:injury group 105.62±17.38 pg/ml control group 56.62±20.57 pg/ml ( P 0.01); NPY:injury group 169.50±95.21 pg/ml control group 46.47±36.41 pg/ml ( P 0.05). Conclusion: SP, CGRP and NPY in injury group were significantly higher than in control group. The results demonstrate that neurogenic inflammatory mediators may play important role in the pathogenic mechanisms of infrapatellar fat pat injury." @default.
- W2384385204 created "2016-06-24" @default.
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- W2384385204 date "2001-01-01" @default.
- W2384385204 modified "2023-09-25" @default.
- W2384385204 title "CHANGES OF NEUROPEPTIDES IN RABBITS INFRAPATELLAR FAT PAD INJURY MODEL" @default.
- W2384385204 hasPublicationYear "2001" @default.
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