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- W2387632128 abstract "It has been well-documented that ICAM-1 implicates in pathophysiology of ischemic-reperfusion injury of the kidney.Renal injury after ischemia appears to be a consequence of tissue hypoxia not only from interrupted blood supply but also from the process of reperfusion which leads to an active inflammatory process.Infiltrating leukocytes are potential source of reactive oxygen species.Proteolytic enzymes and cytokines,which during reperfusion may play a detrimental role.It has been suggested that ICAM-1 plays a key role during leukocyte adhesion and recruitment to inflamed tissue.The goal of the review is to explore the effect and significance of ICAM-1 as well as its mechanism in renal ischemia-reperfunsion." @default.
- W2387632128 created "2016-06-24" @default.
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- W2387632128 date "2007-01-01" @default.
- W2387632128 modified "2023-09-25" @default.
- W2387632128 title "ICAM-1 and renal ischmic-reperfusion injury" @default.
- W2387632128 hasPublicationYear "2007" @default.
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