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- W2389431336 abstract "Objective:To investigate if nitric oxide(NO)will be involved in the cerebral vascular response during cerebral ischemia -reperfusion. Methods; We measured the changes of pial - micrangium' s diameter, NO content and micrangium' s ultrastnicture of the cortex in gerbils by Laser Doppler and electrical chemistry. Results: A linear contraction of the arteriole was observed during initial ischemia. After ischemia for 50 + 10minutes, the arteriole dilated to the maximum value and NO content increased to the maximum, too. Then the micrangium contracted slowly. When ischemia for 2 hours, segmental spasticity of the micrangium was observed and NO content decreased. The endothelial cells of micrangium were severely injured. In the group that reperfusion was performed for Sminutes on the 30th minute of ischemia,NO content increased and the micrangium dilated.In the group that reperfusion was performed for 5 minutes on the 2nd hour of ischemia, NO content decreased and the arteriole contracted. Conclusion: NO content increased in mild cerebral ischemia, which indicated that NO takes part in the cerebral vascular response of dilatation. The endothelial cells of the micrangium in gerbils were severely injured in serious cerebral ischemia and the 02 - NO path was blocked, which indicated that NO takes part in the cerebralstealing bloodresponse." @default.
- W2389431336 created "2016-06-24" @default.
- W2389431336 creator A5032181288 @default.
- W2389431336 date "2003-01-01" @default.
- W2389431336 modified "2023-09-25" @default.
- W2389431336 title "Effects of nitric oxide on cerebral vascular response during cerebral ischemia - reperfusion" @default.
- W2389431336 hasPublicationYear "2003" @default.
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