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- W2391663052 abstract "AIM: To explore the mechanism of lipopolysaccharide(LPS)-induced B7-H1 expression in pancreatic carcinoma cell line Panc-1.METHODS: The levels of phosphorylated p38 mitogen-activated protein kinase(p-p38),phosphorylated extracellular signal-regulated kinase(p-ERK) and phosphorylated c-Jun N-terminal kinase(p-JNK) after stimulated with LPS or treated with mitogen-activated protein kinases(MAPKs) inhibitors were detected by Western blotting.The expression of B7-H1 in Panc-1 cells after LPS stimulation or MAPKs inhibitor treatment was measured by real-time PCR and Western blotting.RESULTS: The levels of B7-H1,p-p38,p-ERK and p-JNK were up-regulated with LPS stimulation.The promoted p-p38,p-ERK and p-JNK levels induced by LPS were inhibited by the corresponding MAPKs inhibitors.Furthermore,the inhibitors of p38 and ERK attenuated LPS-induced B7-H1 expression.However,JNK inhibitor had very little effect on LPS-induced B7-H1 expression.CONCLUSION: LPS induces B7-H1 expression in pancreatic carcinoma cell line Panc-1.ERK and p38 are involved in this regulation as the inhibitors of ERK and p38 attenuate LPS-induced B7-H1 expression." @default.
- W2391663052 created "2016-06-24" @default.
- W2391663052 creator A5086183062 @default.
- W2391663052 date "2011-01-01" @default.
- W2391663052 modified "2023-09-23" @default.
- W2391663052 title "LPS induced B7-H1 expression in pancreatic carcinoma Panc-1 cells" @default.
- W2391663052 hasPublicationYear "2011" @default.
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