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- W2391759811 abstract "Vulvovaginal candidiasis (VVC) is an opportunistic fungal infection caused by Candida species. Local cell-mediated immunity (CMI), rather than systemic CMI, has recently been considered to be the predominant host defense mechanism against vaginal fungi. It appears that coordination of local Th1 and Th2 determines the susceptibility to Candida sepicies. On the other hand, humoral immunity and an innate host defense mechanism against Candida albicans in vaginal epithelial cells may also contribute to resist to VVC. Therapeutic vaccines,antibodies against candida-specific antigens and cytokines may be new immunotherapeutic strategies to control vulvovaginal candidiasis." @default.
- W2391759811 created "2016-06-24" @default.
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- W2391759811 date "2004-01-01" @default.
- W2391759811 modified "2023-09-28" @default.
- W2391759811 title "Immunological research on vulvovaginal candidiasis" @default.
- W2391759811 hasPublicationYear "2004" @default.
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